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Naltrexone blocks opioid receptors in the hypothalamus, removing inhibition of pro-opiomelanocortin (POMC) neurons that regulate satiety

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In fact, carbon-13 nuclear magnetic resonance ( 13 C-NMR) spectroscopy showed that the acetyl moiety of acetyl-l-carnitine was metabolised in both astrocytes and neurons to generate energy [25]

They act on the hypothalamus to mimic satiety

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