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Furthermore, in vivo experiments showed that CNS exhibited therapeutic effects comparable to those of fluoxetine, effectively ameliorating symptoms of anxiety, despair, and anhedonia while also inhibiting microglial activation in the hippocampus of mice subjected to CUMS
In another study, the novel small lipotripeptides DICAMs, which are non-cationic fatty amine-tripeptide conjugates, effectively prevented the formation of biofilms by breaking down biofilms and eliminating the biofilm-affixed bacteria [91]
The mechanism is thought to involve binding of the peptide C terminus to the receptor extracellular domain, followed by insertion of the peptide N terminus into the pocket formed by the transmembrane helices and extracellular loops, instigating receptor activation and signalling 19
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