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group presented that iron overload-induced ferroptosis was a major contributor to doxorubicin (DOX) induced cardiomyopathy, and treatment with the ferroptosis inhibitor ferrostatin-1 remarkably improved the survival of DOX-treated mice
The resultant mice were heterozygous for IGF-1R
doi: 10.1007/s11064-012-0775-4 Chauhan A, Chauhan V, Brown WT, Cohen I
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