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Protection against ROS is probably due to the protective cytokines and other products by ADSCs, such as IGF, which can prevent oxidation damage on fibroblasts
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(6) Increased bone marrow adipose tissue accumulation and altered mesenchymal stem cell differentiation contribute to reduced osteoblast activity, increased osteoclastic resorption, and an overall decline in bone strength, leading to a greater fracture risk despite higher bone mineral density measurements
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