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Toll-like receptor (TLR) response in chikungunya virus infection: mechanism of activation, immune evasion, and use of TLR agonists in vaccine development
The researchers documented severe T-cell hyperproliferation and pronounced mitochondrial lipid oxidative damage, concluding that oxidative stress is a shared mechanistic basis for the debilitating fatigue seen in both conditions
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We conducted immune cell infiltration analysis of the GSE14668 and GSE96851 datasets using CIBERSORTx to further differentiate the distinct immunological microenvironments between the HBV-ALF and HC groups
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