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123,132 In melanoma, BRAFi/MEKi-induced reactivation of MAPK signaling in tumor cells promotes the secretion of CXCL1, HGF, and TGF-, thereby recruiting cancer-associated fibroblasts and immunosuppressive myeloid cells and remodeling the extracellular matrix to generate a drug-resistant microenvironment

Burk et al., 2014) and severe neurological dysfunction upon administration of a Se-deficient diet (Hill et al., 2004

In contrast to EAE where the primary stimulus is immune upregulation in the presence of extrinsic myelin antigens, the CAE model illustrates that MS-like pathology can also be generated by an upstream intrinsic myelin injury, releasing antigens which secondarily trigger white matter inflammation in an immune-predisposed host

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