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Rab7 Overexpression and Drug Candidates Reduce Cholesterol Accumulation in Neuronal SH-SY5Y Cells To date, the ability of upregulated Rab7 activity to bypass NPC1 deficiency and activate alternative cholesterol export pathways has been demonstrated only in NPC1 patient fibroblasts and NPC1 mutant CHO cells [12, 15, 16]
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