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More specifically, the researchers posited that the peptide may have enhanced the expression of Ki-67 and CD98hc that are less intensively synthesized during cell aging. Epithalon also seemed to inhibit the activity of Caspase-3, a crucial enzyme believed to facilitate apoptosis (programmed cell death)
doi: 10.1016/j.ajem.2021.09.033 [PubMed] 37423637 Sharma DS, Gupta S, Sharma P
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