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Lateral gene transfer and parallel evolution in the history of glutathione biosynthesis genes (.) // Genome biology : journal

Therefore, upregulation of GGT in tumor cells provides a potential mechanism to resist oxidative stress and foster tumor progression

When oxidative stress (from inflammation, toxins, or mitochondrial dysfunction) depletes GSH faster than glutathione reductase can regenerate it, the ratio drops, triggering cellular stress responses including NF-kB activation and apoptosis

This pattern reflects the time required for tissue repair processes to manifest as functional improvements

Discontinuation, however, may result in a gradual return to the natural skin tone