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Rosenbaum explains

Where Do FOXO4, p53, and Apoptosis Fit in Pathway Context

GlyNAC (glycine and N-acetylcysteine) supplementation in mice increases length of life by correcting glutathione deficiency, oxidative stress, mitochondrial dysfunction, abnormalities in mitophagy and nutrient sensing, and genomic damage

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Candida produces acetaldehyde, which is structurally similar to formaldehyde and is highly neurotoxic: it depletes glutathione (the brains primary antioxidant), impairs dopamine metabolism, and damages mitochondria in neural tissue