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The intersection between DM and AD further highlights the shared pathophysiological mechanisms, particularly the disruption of insulin signaling pathways in the brain
Suppression of p38 MAPK and JNK via Akt-mediated inhibition of apoptosis signal-regulating kinase 1 constitutes a core component of the beta-cell pro-survival effects of glucose-dependent insulinotropic polypeptide
Tauro JR, Gemeinhart RA: Matrix metalloprotease triggered local delivery of cancer chemotherapeutics
Nausea is common after starting these medications, but this generally ameliorates after 1-2 weeks of therapy
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