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The collagenase model replicates the matrix disorganization and degenerative pathology of clinical tendinopathy more closely than transection models, strengthening the relevance of the findings to the human chronic tendinopathy condition
This deficiency can cause a range of health problems, from fatigue to nerve damage
The primary distinguishing factor is AOD 9604s structural modification for extended half-life and its more extensive research history, including human safety data
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