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Maladaptive repair of TECs leads to the transformation of injured TECs into a secretory phenotype and elicits proinflammatory (e.g., interleukin, tumor necrosis factor, and chemokine) [42,43,44]and profibrotic cytokines [45] (e.g., TGF- and CTGF)
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The addition of antioxidants to the adapted media proves that GSH can enhance the viability of Bali cattle oocytes after vitrification
