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Etiopathogenesis of vitiligo Stress responses in the skin eliciting an autoimmune response in genetically susceptible individuals are thought to be instigated by a trigger event that eventually targets the melanocytes predisposing individuals to develop vitiligo

Studies in diabetic animal models show that antioxidant treatment with N-acetyl cysteine and -tocopherol dramatically reduces oxidative stress, increases antibiotic sensitivity of biofilms, and promotes proper collagen deposition and remodeling

Anti-Inflammatory and Cytoprotective Reduces systemic inflammation markers

Additionally, luteolin 8-C-b-fucopyranoside suppressed secretion of MMP-9, IL-8, ERK/NF-B, and ERK/AP-1 signaling in MCF-7 cancer cells in vitro (156)