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To determine the impact of the relationship between MAP and terlipressin on HRS-AKI reversal: 1) We tested the interaction between MAP and terlipressin in Cox models for HRS-AKI reversal and 2) completed a mediation analysis between terlipressin and time-weighted MAP on full HRS-AKI reversal

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however, the reality is that brain tissue damage often worsens, leading to more severe consequences such as aggravated neurological dysfunction, brain edema, and cell death (Dai et al., 2024b

As a result, a new ferroptosis inhibition axis with GTP cyclohydrolase-1 (GCH1) as the core was finally determined, which was found to be mainly regulated by the GCH1/BH4/DHFR pathway, which also protected cells from ferroptosis in a GPX4-independent manner (49, 50) ( Figure 1D )