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Table 3 summarizes the main triggers and mechanisms associated with eryptosis across various clinical conditions
The resulting decrease in p62 leads to reduced sequestration of Keap1, thereby increasing Nrf2 stability and activating transcription of anti-ferroptosis genes [110]
Collectively, these findings underscore IFs potential as a modulator of GHIGF-1 signaling and indicate that there should be further research into its physiological and therapeutic applications
UCP1 transcript abundance does not necessarily predict protein abundance, and protein abundance alone does not prove increased thermogenic flux
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