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ROS derived from NOX family enzymes are inherently dependent on NADPH produced by G6PD while mitochondrial ROS, for example, are largely NADPH-independent

Since the distributions of GLP-1R in sino-atrial node and ventricular cardiomyocytes [31, 38], a previous study indicated that the direct activations of GLP-1R effectively opposed the effects of -adrenoceptor stimulation on cardiac ventricular excitability and might be responsible for HR augmentation or arrhythmias [39]