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Through NRF2-driven GSH synthesis, tumor cells are protected from oxidative stress, which contributes to chemotherapy resistance by reducing the cytotoxic effects of ROS generated during treatment
As a result, the activity of the electron transport chain (ETC) is hindered, decreasing ATP production during oxidative phosphorylation [8]
Because the heart relies heavily on fatty acids as an energy source, efficient fatty acid transport plays an important role in maintaining normal cardiac energy metabolism
Multiple and additive functions of ALDH3A1 and ALDH1A1: cataract phenotype and ocular oxidative damage in Aldh3a1(/)/Aldh1a1(/) knock-out mice
