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The compound has been found to operate in the postrema and nucleus of the hindbrain solitary tract in order to both induce satiety and regulate appetite by acting on receptors found in the laterodorsal tegmental nucleus, ventral tegmental area, and the hypothalamus [1]
Effect of riboflavin deficiency on development of the cerebral cortex in Slc52a3 knockout mice
A 12-week clinical trial showed subjects taking AOD-9604 (1 mg/d) lost an average of 2.6 kg, compared to 0.8 kg in the placebo group.
AOD-9604 administers subcutaneously, meaning into the fat layer just beneath the skin
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