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doi: 10.1021/acsnano.4c04901 Summary Keywords diabetic cardiomyopathy, ferroptosis, mitochondrial damage, molecular mechanisms, targeted therapy, combination therapy, clinical applications Citation Liu B, Jin Q, Sun YK, Yang ZM, Meng P, Zhang X, Chen Q, Gan P, Zhao T, He JJ, He GP and Xue Q (2025) From bench to bedside: targeting ferroptosis and mitochondrial damage in the treatment of diabetic cardiomyopathy

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As we age, NAD+ levels naturally decline, which may contribute to fatigue, reduced mental clarity, and slower recovery