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Vascular endothelial growth factor (VEGF) is a major factor in embryonic development and regulation of physiological angiogenesis for reproductive function

Semaglutide's activation of these receptors can modulate neurotransmitters like serotonin and GABA, creating variable effects depending on individual brain chemistry

Consequently, this review examines the hypothesis that semaglutide might primarily serve as a prevention-oriented neuroprotective intervention rather than a disease-modifying therapy for symptomatic Alzheimers disease

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