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Altogether, these results demonstrate that CNS GIPR antagonism leads to body weight loss, and both central and systemic dosing of mGIPR-Ab can target the CNS to regulate body weight
Therefore, this review aims to synthesize the evidence on the mechanistic expansion of GLP-1RAs from metabolic regulators to systemic modulators of inflammation, autophagy, and organ protection, and explores their therapeutic repurposing across diseases
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