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Existing evidence indicates that eating disorders can increase the risk of diabetes, obesity, hypertension, hypertriglyceridemia, headache, back pain, and other chronic metabolic diseases

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5b), and genome-wide significant associations between SPHKAP gene variants, T2D risk, and high BMI have been found in meta-analyses from four separate genome-wide association studies (GWAS) in humans 37, 38, 39, 40 (Supplementary Fig

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Coordinate regulation of glutathione biosynthesis and release by Nrf2-expressing glia potently protects neurons from oxidative stress